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Following the Wessells 1998/2000 ED studies establishing MT-2’s central erectogenic biology, the primary limitations for clinical development — potent MC1R-driven tanning (unsuitable for a sexual dysfunction drug requiring on-demand dosing), short plasma half-life, and prominent nausea — motivated the identification of PT-141 as the primary active CNS metabolite of MT-2 (the deaminated C-terminal carboxylate form) with reduced MC1R activity and preserved MC4R/MC3R CNS biology